Reduces the effectiveness of uricosuric drugs. Concomitant use of high doses of paracetamol enhances the anticoagulant effect of drugs (reduction of procoagulant factors synthesis in the liver).
Ethanol contributes to the development of acute pancreatitis.
Long-term trenbolone acetate cycle sharing of paracetamol and other nonsteroidal anti-inflammatory drugs increase the risk of “analgesic” nephropathy and renal papillary necrosis, onset of end-stage renal failure.
Diflunisal increases the plasma concentration of paracetamol by 50% – the risk of hepatotoxicity.
Myelotoxicity drugs increase the expression gematotoksichnosti drug.
Caffeine: when combined with caffeine cimetidine, oral contraceptives, ciprofloxacin, norfloxacin – reduction of caffeine metabolism in the liver (slowing down its clearance and increase in the blood concentration). Mexiletine – reduces the excretion of caffeine and 50%; nicotine increases the rate of caffeine removal.
Monoamine oxidase inhibitors, furazolidone, procarbazine, and selegiline – large doses of caffeine can cause the development of dangerous cardiac arrhythmias or pronounced increase in blood pressure.
It accelerates the absorption and enhances the action of cardiac glycosides increases their toxicity.
The combined use of caffeine with beta-blockers may lead to suppression of mutual therapeutic effects; with adrenergic bronchodilators drugs – for extra stimulation of the trenbolone acetate cycle central nervous system and other additive toxic effects.
Caffeine may reduce theophylline clearance and possibly other xanthines, increasing the possibility of additive pharmacodynamic and toxicity.
Ascorbic acid: increases the concentration in the blood penicillin and tetracycline. At a dose of 1 g / day increases the bioavailability of ethinyl estradiol (including a member of the oral contraceptive).
Reduces the effectiveness of heparin and indirect anticoagulants. Improves vsasshanie in the intestine of iron supplementation (translates ferric to ferrous); may increase the excretion of iron while the use of deferoxamine.
Acetylsalicylic acid (ASA), oral contraceptives, fresh juices and alkaline water reduces vsasshanie and assimilation of ascorbic acid. ‘
In an application with ASA increased urinary excretion of ascorbic acid and reduced excretion of ASA. ASA reduces absorption of ascorbic acid is about 30%.
It increases the risk of crystalluria salicylates in the treatment of sulfonamides and short-acting, slow excretion by the kidneys acids, increases the excretion of drugs having an alkaline reaction (including alkaloids), reduces blood levels of oral contraceptives.
In an application reduces the chronotropic effect of isoprenaline.
Calcium gluconate: while the use of quinidine may slow intraventricular conduction and increase quinidine toxicity.
It forms insoluble complexes with the antibiotic tetracycline (reduces the antibacterial effect).
It slows down the trenbolone acetate cycle absorption of tetracyclines, digoxin, oral iron supplementation (between their methods of interval should be not less than 2 hours).
In combination with thiazide diuretics can exacerbate hypercalcaemia, reduce the effect of calcitonin hypercalcemia. It reduces bioavailability of phenytoin.
Pharmaceutical incompatible with carbonates, salicylates, sulfates (forms insoluble or poorly soluble calcium salt)., Reduces the blockers “slow” calcium channels.
Rutoside: pharmacological effect is enhanced by ascorbic acid.
In order to prevent liver toxicity should not combine the drug with the use of alcoholic beverages.
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